Combination of herbs that:
• Inhibits the COX-2 enzyme and reduces inflammation*
• Promotes healthy joint function*
• Promotes normal cell growth*
• Contains organic herbal anti-aging constituents that inactivate free-radicals*

Per the company;
New Chapter has extensively researched the herbal pharmacopoeia and international medical databases and discovered that the following time-tested herbs, properly extracted and blended in the correct proportions, contain at least 8 phytonutrients that may safely and significantly inhibit COX-2:*
HOLY BASIL (Ocimum sanctum) Contains the powerful COX-2 inhibitor ursolic acid, which significantly enhances detoxification and reduces inflammation.*
TURMERIC Unique curcumin phytonutrient complex, naturally inhibits inflammatory COX-2; synergistic with green tea, significantly multiplying anti-inflammatory effect of green tea polyphenols.*
GINGER Supercritical extract inhibits both inflammatory COX and 5-LO and offers numerous anti-aging constituents.*
GREEN TEA Proceedings of the National Academy of Sciences report green tea polyphenols markedly reduce COX-2.* Major university database notes green tea contains 51 anti-inflammatory phytonutrients.*
ROSEMARY Dual extracts offer highly concentrated, full spectrum, COX-2 inhibition and support detoxification.*
HU ZHANG (Polygonum cuspidatum) Richest known resveratrol source, shown scientifically to inhibit inflammatory COX-2.*
CHINESE GOLDTHREAD AND BARBERRY Unique berberine phytonutrient complex, naturally inhibits inflammatory COX-2.*
OREGANO Source of large number of anti-inflammatory compounds (31) according to USDA database.*
SCUTELLARIA Unique baicalin phytonutrient complex, naturally inhibits inflammatory COX-2.*
*This statement has not been evaluated by the Food and Drug Administration. This product is not intended to diagnose, treat, cure or prevent any disease.

Research on Zyflamend:

Effects on Prostate Cancer Cells

Another study showing decreased proliferation of prostate cancer cells

Preliminary Trial in Prostate Cancer Patients

Effects on Gene Expression in NF-kappa B